LFT Interpreter

Pattern recognition & investigation guidance for Australian GP

Aligned with GESA 2024 · RCPA · RACGP
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Reference
Patient Details
Ageyears (needed for FIB-4)
Alcoholstandard drinks / week
Liver Enzymes
ALTULN 40 U/L
ASTULN 40 U/L
ALPULN 130 U/L
GGTULN 60 U/L
Bilirubin & Synthetic Function
Bilirubin (total)ULN 20 µmol/L
Albumin35–50 g/L
Fibrosis Risk — FIB-4 (GESA 2024)
Platelets× 10⁹/L

FIB-4 = (Age × AST) ÷ (Platelets × √ALT)
Validated for age ≥ 35. GESA recommends as first-line fibrosis assessment in MAFLD.

Clinical decision support only — does not replace clinical judgement.
Aligned with GESA MAFLD Consensus Statement 2024, RCPA FIB-4 Implementation Guide, and RACGP resources.
Reference ranges may vary between laboratories — check local values.

GESA MAFLD Consensus 2024 · RCPA FIB-4 Guide
Patterns of LFT Derangement
PatternKey Features
HepatocellularALT/AST ↑↑ predominant (R-factor ≥ 5). Viral, toxic, autoimmune, metabolic.
CholestaticALP ↑↑ predominant (R-factor ≤ 2). Obstruction, PBC, PSC, drug-induced.
MixedR-factor 2–5. Drug reactions, granulomatous, evolving disease.
Isolated GGT ↑Non-specific. Alcohol, enzyme inducers, MAFLD, obesity.
Isolated ALP ↑If GGT normal → bone origin. If GGT ↑ → hepatobiliary.
Isolated bilirubin ↑Gilbert's if unconjugated, mild, LFTs otherwise normal.
R-Factor (ACG Method)

R = (ALT ÷ ALT ULN) ÷ (ALP ÷ ALP ULN)

R ≥ 5 → Hepatocellular
R ≤ 2 → Cholestatic
R 2–5 → Mixed

De Ritis Ratio (AST : ALT)
RatioSuggests
< 1 (ALT > AST)Viral hepatitis, MAFLD, acute toxic injury
> 1Alcohol, advanced fibrosis/cirrhosis, muscle
> 2 (AST < 300)Strongly suggests alcohol-related liver disease
FIB-4 — GESA 2024 Cutoffs
ScoreRiskAction
< 1.3LowRepeat FIB-4 every 1–3 years
1.3 – 2.67IndeterminateSecond-line: Fibroscan (VCTE), ELF, or Hepascore
> 2.67HighRefer to hepatology / liver clinic

Age < 35: FIB-4 less validated — interpret with caution.
Age ≥ 65: Consider adjusted lower cutoff of 2.0 (lower specificity at standard cutoff).
Second-line thresholds: VCTE ≥ 8 kPa · ELF ≥ 9.8 · Hepascore ≥ 0.60 → refer to specialist.

Non-Invasive Liver Screen (NILS)
Viral: HBsAg, HCV Ab, (HEV IgG if acute presentation)
Autoimmune: ANA, SMA, anti-LKM, Immunoglobulins (IgG, IgA, IgM)
Iron: Ferritin + transferrin saturation
Metabolic: Caeruloplasmin (if age < 40), alpha-1-antitrypsin
Metabolic syndrome: Fasting glucose or HbA1c, fasting lipids, BMI, waist circumference
Other: Coeliac screen (tTG-IgA), TFTs, CK (if AST ↑ to exclude muscle)
Imaging: Liver ultrasound
Fibrosis: FIB-4 ± Fibroscan / ELF / Hepascore
Urgent Referral Criteria
Refer urgently / same-day if any of:
• ALT > 500 U/L
• Bilirubin > 100 µmol/L
• Clinical jaundice with deranged LFTs
• Features of decompensated cirrhosis (ascites, encephalopathy, variceal bleeding)
• Suspected paracetamol toxicity
• Imaging suggestive of hepato-pancreato-biliary malignancy
When to Repeat LFTs
SeverityRepeat Interval
NormalNo routine repeat needed
Borderline (1–1.5× ULN)3–6 months (sooner if clear transient cause identified)
Mild (1.5–3× ULN)4–6 weeks to confirm persistence
Moderate (3–10× ULN)4–6 weeks alongside investigation
Severe (10–25× ULN)1–2 weeks with urgent workup
Massive / Urgent (>25× / >500)24–72 hours / daily if admitted
Isolated GGT ↑3–6 months (address cause first)
Isolated bilirubin ↑ (Gilbert's)No routine repeat needed
Isolated ALP ↑ (bone)As clinically indicated for bone condition

Key principle: Confirm persistence before committing to a full workup. 16–30% of mildly elevated LFTs normalise on repeat testing. If normalised → likely transient cause → reassure.
Post-investigation: If cause identified and managed (e.g. MAFLD with lifestyle change, medication stopped), repeat in 3–6 months to confirm improvement. If FIB-4 low risk, repeat FIB-4 every 1–3 years (GESA 2024).

MAFLD Diagnostic Criteria (GESA 2024)
Hepatic steatosis (on imaging or biopsy) PLUS at least one of:
• BMI ≥ 25 kg/m² (or ≥ 23 in Asian ethnicity)
• Type 2 diabetes
• ≥ 2 metabolic risk factors (waist circumference ↑, hypertension, triglycerides ↑, HDL ↓, prediabetes, insulin resistance, CRP ↑)